Biomedical Treatment Out of Control by Michael R. Valentine, Ph.D.
Labels as Perceptions of Reality Is it a picture of flies stuck to a Shell Pest-Strip? A window with curtains half open, revealing snowflakes or rain outside? Germs under a microscope? People seen from the top of the Empire State Building? A partially open observatory dome showing the stars? Although the lines of the drawing never change, one begins to perceive them differently once a word, a label, a trait, a concept, or a belief is attached. From what I understand, this picture is actually a drawing in a museum entitled, "A giraffe passing a second-story window." In response to the suggestion that the drawing is a giraffes neck passing a second-story window, one sees it as a giraffe. One begins to see it, to believe it, and to react and feel toward it as if it were really a giraffe. This fundamental principle-that the words used to define a concept have profound effects on the way the world is seen-is one of the critical problems in education in general, the whole area of learning disabilities, and the concept of attention deficit/hyperactivity disorder, specifically. Schools Plus the Medical Model Equals the Diagnosis of Kids In the past thirty years, the medical model has infiltrated psychology and educational institutions in a big way. The Diagnostic and Statistical Manual (DSM), the psychiatric bible of labeling individuals, has grown from about 60 disorders in its first addition to over 300 disorders in the fourth edition. Almost all of these disorders, largely created by committee, have fallen prey to the medical model's assumption that any deviations or variations from "normal behavior" are "caused" by genetics or a biochemical imbalance. In recent years almost every behavioral phenomenon has moved from a psychological, educational, learned or choice perspective to a medical model, genetic, biochemical imbalance determined perspective. Fears, anxiety, phobias, compulsions, eating disorders, learning disabilities, ADHD, alcoholism, etc. are just a few of the now popularly believed medical disorders. I've also read reports in which the medical field is trying to link violence, criminality, homosexuality, and even marital infidelity to genetic and biochemical imbalances. Most disheartening to me is that within two to three decades proponents of the medical model have created a delusional reality that millions of American children, mainly males, are biochemically imbalanced, neurologically defective, genetically flawed rejects, and, in some shape or form, handicapped--regardless of the fact there is no hard scientific evidence to substantiate their position. Some years back, Stanton Peele wrote a book, The Diseasing of America: Addiction Treatment Out of Control. The main thesis of the book, which dealt with how various addictions, such as alcoholism, moved from a cultural, behavioral, characterlogical problem to a very profitable medical "disease", could be applied to various educational difficulties and the book could be retitled, The Diseasing of American Children: Biomedical Treatment Out of Control in Our Schools. A good example of the magnitude of the shift in education from a cultural, learned behavioral perspective to a medical modal perspective would be the assumed "biochemical disorder" of attention deficit disorder (ADD), or attention deficit/hyperactivity disorder, (ADHD). If this biochemical disorder were true, then within a normal distribution curve of all children there would be a "magical diagnostic" line that would separate normal biochemistry from abnormal biochemistry.
The ultimate question is, is there any test or assessment instruments that can determine this biochemical imbalance, let alone the magic line that differentiates the groups? The answer emphatically and absolutely is no. If you have doubts about what I say, I challenge you to critically reread the literature on AD/HD. As a way to challenge your thinking about this literature and, I hope, see there is no substantiated evidence for this medical model perspective, I give you two examples, out of hundreds which say the same thing, that there is no proven evidence for a biochemical imbalance. The first is taken from School Psychology Review - VOLUME 20, NO.2, 1991. This particular section of the journal was designed to inform us about the "Neurobiological Basis of ADHD" and was written by George Hynd, Kelly Hern, Kytja Voeller, Richard Marshall. I have underlined words or phrases which will point out the speculative and unsubstantiated position of their argument. ... p. 178 Neuroanatomical Basis of ADHD Table 1 lists a number of neuroanatomically-based hypotheses about the brain regions that may be involved in ADHD. These same regions are rich in catecholamines. Generally, these hypotheses implicate subcortical structures believed important in arousal and in the regulation of motor control (Laufer, Denhoff, & Solomons, 1957; Slatterfield & Dawson, 1971) or they propose the involvement of both subcortical and cortico-frontal systems (Dykman, Ackerman, Clements, & Peters, 1971; mattes, 1980; Voeller & Heilman, 1988; Hynd, Semrud-Clikeman, Lorys, Novey, & Eliopulos, 1990). Laufer, Denhoff, and Solomons (1957) also proposed that midbrain structures (thalamus, hypothalamus) may be involved in ADHD. However, the specific structures and neurotransmitters involved, especially in the thalamus, are largely unknown and research into the contributions of hormones related to the hypothalamus has not been fruitful. Numerous studies implicate the Reticular Activating System (RAS), a system of nerve cells and brain stem structures extending into the midbrain. The idea that lowered levels of RAS functioning might be implicated in ADHD (Satterfield & Dawson, 1971), or that limbic structures might be dysfunctional, has encouraged a great deal of research, but most results are inconclusive. The role of the RAS in arousal and its extensive connections with the frontal lobes continues to suggest RAS involvements in ADHD. However, investigative methodologies employed in examining subcortical structures and their functional systems have limited our ability to adequately validate some of these theories. Neuroimaging Studies p. 181 It is relevant to note that in none of the neuroimaging studies have the brain scans been read a clinically abnormal; the brains of children with ADHD look normal.... As suggested by the imaging and postmortem studies of dyslexics, these differences (what differences? There were none.) may be related to prenatal deviations in cellular migration and maturation (Geschwind & Galaburda, 1985a; 1986b; 1985c; Hynd & Semrud-Clikeman, 1989). Note particularly the last paragraph in which the authors say, "It is relevant to note that none....", yet their conclusion some how suggests a neurobiological basis for ADHD. As a second example, look at the 1991 Zametkin study that has been cited by CHADD (The parent organization for Children with ADHD), Barkley (a leading expert on ADHD) and Time Magazine as proof that there is a biochemical basis for this disorder. Listed below are some excerpts from the original study. Again, I have underlined the speculative words and phrases and have made some comments which are italicized. The New England Journal of Medicine, November 15, 1990, Vol. 232, Number 20 CEREBRAL GLUCOSE METABOLISM IN ADULTS WITH HYPERACTITIVY OF CHILDHOOD ONSET Zametikn, Nordahl, Gross, King, Semple, Rumsey, Hamburger, & Cohen (Comments in parentheses are mine) ...The cause of childhood hyperactivity is unknown. ...Hyperactivity is a disorder of unknown cause. Despite extensive efforts, researchers have been unable to demonstrate consistent neurobiologic differences between hyperactive children and normal controls. Therefore, the validity of hyperactivity as a syndrome remains controversial, as does its treatment with stimulant medication. ...Adults were chosen as subjects because of ethical concerns about exposing children to a radioactive tracer. (Where are the ethical concerns about exposing millions of children to a highly addictive Class II drug with no proven long term beneficial outcomes?) ...no consistent finding has emerged to implicate a specific neurotransmitter, modulator, or neuroanatomical substrate. ...Earlier neuroanatomical imaging studies of patients with attention deficit disorder with hyperactivity either have found no abnormality or have included large numbers of subjects with substance-abuse disorder, making it difficult to determine whether the findings were secondary to chronic drug use or part of the syndrome. In addition, in the studies by Lou et al. of cerebral blood-flow abnormalities in attention deficit disorder with hyperactivity, there was a large discrepancy in age between the patients and controls, and the study included subjects with developmental dysphasia and mental retardation. (By the way, go back to School Psychology Review, Vol. 20 pp. 178-181 and see if they told you that the Lou, et. al. study was done on thirteen subjects with developmental dysphasia, mental retardation and/or had been on medication for long extended periods of time.) ...Sample: ...normal adults (28 men and 22 women; mean age 36.3+11.7 years). The 25 patients (18 men and 7 women) (Note the difference in the women sample size) ... they reported a definite childhood history of attention deficit disorder with hyperactivity. (Self reporting is probably not a very reliable indication of whether a person has a disease.) ...Findings: ...global cerebral glucose metabolism was 8.1 percent lower in the hyperactive patients than in the controls. (What does a 8.1 glucose metabolism difference really mean? Does this difference prevent a student from raising his hand, hitting someone, talking back to the teacher, etc? Check out the research on glucose metabolism and see how it varies by intelligence; level of mastery of any particular skill.) ...The results for men only were similar ... but the difference was not significant. When global cerebral glucose metabolism was compared in the 7 hyperactive women and the 22 normal women, it was found to be 12.7 percent lower in the hyperactive women (9.02+1.29 mg per minute per 100 g vs. 10.33+2.07 mg per minute per 100 g), but the difference was not significant. (Even with a small 8.1% difference in glucose metabolism; the differences disappear when males are compared to males and females to females only. Remember the small number of women in the experimental group. Could this mean the difference, if any, might be sex linked?) ...The finding that 4 of 60 regions had significantly depressed normalized metabolic rates could be explained by chance alone and thus requires replication. (There was a replication study done this time on adolescents that showed there were no significant differences between groups or between males or females. CHADD did not send this information all over the country to advertise there were no differences.) ...The patients and controls did not perform differently on the continuous-performance test. (Even with 8.1% glucose differences there were no differences in outcome measures or performance variables.) ...We feel confident that neither the small number of women in our sample nor the presence of patients with specific reading or arithmetic disabilities accounts for the differences we observed between patients and controls. These two examples from the literature are typical. Their conclusions sound convincing, but on closer examination, they have no evidence to substantiate their conclusions, yet many people assume a case for biochemical causes of ADHD has been made. Witness some of following news releases. (My underlines for emphasis.): BRAIN FUNCTION YIELDS CLUES TO HYPERACTIVITY Sally Squires-Washington Post 11/15/90 The report (Zametkin's study) "substantiates the notion that this is a biological condition..." - Joseph Biederman, Mass. Gen. Hosp. FOR SOME FAMILIES, HYPERACTIVITY STUDY IS A RELIEF Sally Squires Medical News-Washington Post 11/20/90 ...NIMH researchers offered the first substantive evidence that hyperactivity is not just a psychological problem, but appears to have a biological basis... ...Diagnosis remains a problem. Doctors still have no good test for the condition and must rely on interviews with parents, teachers and the affected child. - Joseph Biederman, Mass. Gen. Hosp. San Jose Mercury News, November 15, 1990 STUDY TIES HYPERACTIVITY TO DISORDER OF THE BRAIN - Robert Engelman "There are people who say you should not use medications, that it is a matter of upbringing," Zametkin said. "We're hoping that this (study) will put an end to that kind of thinking." (The Zametkin study , which had no definitive conclusions, is used by this author to justify medicating millions of children and stop any critical thinking that there might be another choice.) The Zametkin study, which really showed nothing and proved nothing, found the front pages of the Washington Post, New York Times, and other major newspapers, with four major television networks all carrying the same story on November 15, 1990. That was the day before Congress voted on whether ADHD should become a handicapping condition under Individuals with Disabilities Education Act (IDEA). I personally find it interesting that four years later, just before Congress was slated to begin procedures for reauthorization of IDEAS, Time Magazine, July, 1994, dedicated its cover story to ADHD. As evidence of this disorder they showed the same pictures of the "no differences in the brains" that Zemetkin found in 1990. Despite the fact that ADHD has been extensively studied, there is no biochemical or medical evidence to determine this disorder. If leading medical or psychological experts on ADHD were given the biochemistry, the PET scans, the MRI's, the evoke potentials, genetic information, or any other medical information from children with ADHD and an equal number of children that were considered normal, could they tell you which group was which? The answer is no, they cannot. Yet, pro ADHD groups do not even question the medical bias and because of that millions of children have been labeled as biochemical-genetically flawed individuals. Most estimates of the incidence of this supposed disorder are in the 3 to 5% range. I find it interesting however, that in a recent report submitted to the Department of Education, so they could have accurate information to help make policy, Shaywitz and Shaywitz estimated that from 10 to 20 percent of all children are ADHD. There are roughly 66 million school aged children in America. Ten to twenty percent equals 6.6 million to 13.2 million children that are supposedly ADHD, and biomedically flawed, according to Shaywitz and Shaywitz. All of this is based on an unsubstantiated medical assumption.
SCHOOL PSYCHOLOGY REVIEW - VOLUME 20, NO.2, 1991, THE NATURE AND CHARACTERISTICS OF ATTENTION-DEFFICIT HYPERACTIVITY DISORDER by Paul Frick & Benjamin Lahey p. 189 ... The differential validity of the current diagnostic construct of ADHD remains to be evaluated empirically. p. 193 To reduce the number of false positives, professionals are now calling for a best estimate approach to diagnosis in child psychopathology. (Or is that a best guess approach?) ...Currently, there is no consensus as to which best estimate procedure to use, ...However, different types of informants (e.g., parent and teacher) can be expected to demonstrate an agreement level no better than approximately 0.30. How, then, should information from different sources be weighed in making best estimate diagnoses? Discordance does not necessarily imply that one informant is right and the other is wrong. (I love professional double talk.) Instead, it may reflect differences in the child's behavior due to particular settings; that is it may reflect some degree of situational specificity. (Even if the informants don't agree, apparently a child may still have the disease.) Exceptional Children, Vol. 60, No. 2, pp. 125-131 EDUCATIONAL ASSESSMENT OF STUDENTS WITH ATTENTION DEFICIT DISORDER - McKinney, Montague, Hocutt ... existing literature on ADD is not adequate as a guide to what assessment data are necessary and sufficient to qualify a child with ADD... Washington Post - 11/20/90 FOR SOME FAMILIES, HYPERACTIVITY STUDY IS A RELIEF ...Diagnosis remains a problem. Doctors still have no good test for the condition and must rely on interviews with parents, teachers and the affected child. Ladies Home Journal - September 1990 WHY CAN'T YOUR HUSBAND SIT STILL? "... the good news is that once diagnosed--through a detailed personal history -- ADD is treatable, either with stimulants such as Ritalin or antidepressants..."
If ADHD is biological, I assume that roughly equal numbers of children in different countries, states and zip codes within those states should have the disorder.
The use of Ritalin by state within the United States is also interesting. According to the Drug Enforcement Agency, in 1991 the top states in the use of Ritalin in grams controlled per 100,000 population were: 1. Idaho - 1401.25; 2. Michigan, 1206.09; 3. Utah, 1116.98; 4. Ohio, 1114.19; 5. Iowa, 1073.49; all the way to the low usage of Hawaii, 338.62 and Maine, 283.61. Within these states the increased difference in grams of methylphenidate used from 1990 to 1991 by specific three digit zip codes (see Fig. IV) show some of the top locations in this country to have the disorder:
It is interesting to note that the change in one year within any one of these three digit zip codes represents more Ritalin used than the total change in some states or even more Ritalin used than most countries in the world. In the US, school psychologists and principals have told me that they can have almost any child walk into certain doctor's offices, clinics or hospitals and know the child will come out with a diagnosis of ADHD. In Louisiana recently, some principals told me that since ADHD has become recognized by law in their state, the school staff spends anywhere from an hour to two and a half hours a day just handing out the medications, where two years prior to this they could count the number of kids on medication in the entire school on one hand. A Colleague of mine asked a psychiatrist what basic information he needed to obtain from the school and the parents regarding what they had done to change the child's behavior, instruction, curriculum, and/or home or school environment, and how long were these changes in place and evaluated before he considered a child ADHD and put him on medications? Based on the nature of this question one might wonder what basic research has been done with regard to educational, and family interventions; what priority has been given by the Department of Education to researching these interventions rather than just accepting the medical models assumption that this is a biochemical genetically caused disorder. The Chesapeake Institute report to the federal government entitled "Executive Summaries of Research Synthesis on the Assessment and Identification of Attention Deficit Disorder" by James McKinney, Marjorie Montague and Anne Hocutt indicated, of the 1,300 articles reviewed which were relevant to assessment and identification of children and youth with ADD, the research on educational characteristics was "limited." "Only about 90 articles even reported education characteristics" and of those 90 articles there was "very little replication of studies." (Does that mean at least one or two were replicated?) If you systematically investigate this subject, not only are there "very few" studies on any relevant educational or family variables, but few if any are well designed longitudinal studies on any outcome measures either educational or medical. See if you can find any definitive data on just basic demographic information -- socioeconomic status, racial or ethnic backgrounds--for this supposed biological disorder. Why isn't this information available? This is the most studied disorder of childhood. There have been over 10,000 studies done on ADHD, yet there are no basic demographics, let alone well-designed longitudinal studies on any meaningful educational or family outcomes. Why not? From what little demographic data is available however, the trend seems to be an increase labeling of the poor, lower SES students and especially minority Black and Hispanic males. This disorder which varies so widely by country, state and zip code, which has no physical markers, no agreed upon definition, no agreed upon assessment and no relevant basic research, also has the unique ability to vary by social contexts. Dr. Russell Barkley, one of the supposed leading experts in the ADHD field, has outlined eight situational task factors that have been observed to affect symptom severity in ADHD, (i.e. one-to-one versus group situations, fathers versus mothers, novelty versus familiarity of the setting or task, etc.) It would be interesting to see how the experts explain how this biochemical disorder knows to turn off the symptoms when Dad walks into the room. At a large four state convention in Kansas for teachers of the emotionally disturbed, one of the major key-note presenters, a psychiatrist, used his hands and fingers to indicate how the neurons worked. He wiggled the fingers on his right hand to indicate the firing of the neuron; used the space between his hands as the neuro-synoptic junction between the dendrites and axons that the neurotransmitters had to cross over; and used his left hand as the receptors of these neurotransmitters which when received properly would then fire up his left fingers so they would wiggle. He then indicated that because of some type of biochemical imbalance the wiggle finger right hand impulses could not be transmitted across the space and make the fingers on the left hand wiggle which, if this were to happen, would somehow inhibit the child's inappropriate behavior. But, he said if we "gave him the gift of Ritalin" it would rectify the imbalance of the neurotransmitters and allow the right hand wiggle finger impulses to cross over the space and start firing the fingers on the left hand receptors, which then "of course" inhibits the child's inappropriate behavior. Combining these two lines of thinking perhaps we should give the child a different gift besides medication. Perhaps we should give those poor imbalanced neurotransmitters the "gift of Dad's" so those left fingers can wiggle to inhibit the symptoms. The proponents of the medical position draw analogies between ADHD and other real diseases, such as diabetes. The symptoms of other real diseases do not change as far as I know when Dad walks into the room, nor do they change if the child is examined one on one or in a group. If a child's symptoms vary based on social contexts, it would appear that the child has some degree of control over his behavior; has learned when or when not to behavior; and could be taught to control his behavior in other situations. If a child can control his behavior in church, when dad is around, in third period when the principal walks into class, in fourth period because he likes the teacher, or when a teacher or adult takes a firm stand and stops the child's inappropriate behavior -- then this child is capable of controlling his behavior and can be taught how to control his behaviors in other situations. Why? Because he has already demonstrated to you that he has "learned" to control those little neurotransmitters in "various situations." We need to get back to educating and training children rather than medicating them. The picture of the giraffe passing the second story window graphically illustrates how one interprets the world is based on the beliefs held about the world. The picture (the lines and dots), or the facts (the child's behavior) are the same. The interpretation of the facts depends whether one has a medical or an educational bias. If it is a medical bias, the child is seen as incapable, diseased, and a flawed individual-someone who can't control his behavior. If there is an educational bias, the child is seen as capable and in the on-going developmental process of learning to control his behavior. There is usually overwhelming evidence that the child is capable and can control his behavior. The intervention strategy from this more hopeful and optimistic perspective is obvious and simple to understand, but often times difficult and time consuming to do. It requires adults to continue to teach, educate and expect children to behave and learn to be responsible for their behavior, and actions. In my opinion, this nation better stop believing that there is a quick medical fix to all our social, academic and behavior problems by giving our children a pill, and begin, instead, to take responsibility for our children in an ongoing, caring, loving, involved, committed, developmentally appropriate long term basis by educating, parenting, teaching, training and disciplining our children -- and hopefully teaching them well. If you are interested in more information on the biomedicalization of education and psychology in general and ADHD specifically, I suggest you read the references in the Bibliography for Alternative Views on ADHD and/or order either my video tape on The Myths of ADHD and/or Dr. Breggin's videos and books on ADHD. (www.breggin.com) |